Flaviviruses — classification?
Positive-sense ssRNA viruses in Flaviviridae.
Flavivirus genome — type?
Non-segmented, single polyprotein RNA.
Flavivirus replication — location?
Cytoplasm within ER-derived vesicles.
DENV vectors — primary?
Aedes aegypti and albopictus mosquitoes.
Zika manifestations — congenital?
Microcephaly and congenital Zika syndrome.
Flavivirus entry — mechanism?
Receptor-mediated endocytosis and fusion.
Genome translation — site?
At the ER as a polyprotein.
Viral maturation — process?
prM cleavage in Golgi.
Dengue severity — key factor?
Prior infection and immune enhancement.
Zika — neurological complication?
Guillain-Barré syndrome.
Picornaviruses — classification?
(+): ssRNA, non-enveloped, T=3 capsid.
Poliovirus vaccine — type?
Inactivated (Salk) and live attenuated (Sabin).
Salk vaccine — characteristic?
Inactivated, no reversion risk.
Sabin vaccine — advantage?
Oral, induces intestinal immunity.
Vaccine reversion — risk?
Sabin vaccine can mutate back to neurovirulent form.
cVDPV — meaning?
Vaccine-derived poliovirus from mutated Sabin strains.
ADE — stands for?
Antibody-dependent enhancement.
ADE — occurs when?
At intermediate cross-reactive antibody titers.
Dengue — severe form?
DHF and DSS with vascular leakage.
NS1 — role in pathogenesis?
Induces endothelial dysfunction and leakage.
Zika — transmission?
Mosquito-borne and vertical (mother to fetus).
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1. What does Flavivirus classification refer to?
2. Which mosquito species are primarily responsible for transmitting flaviviruses such as Dengue and Zika?
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