Flashcards : Cell Injury and Death Mechanisms — 24 cartes

Toutes les cartes

1Question

Homeostasis — definition?

Réponse

Steady functional state of a cell.

2Question

Adaptation — role?

Réponse

Reversible cellular changes to survive stress.

3Question

Cell injury — triggers?

Réponse

Exceeds adaptive limits, nutrient deficiency, or function impairment.

4Question

Reversible injury — hallmark?

Réponse

Cell swelling and hydropic change.

5Question

Irreversible injury — key feature?

Réponse

Severe membrane damage and mitochondrial failure.

6Question

Necrosis pattern — coagulative?

Réponse

Architecture preserved, seen in infarcts.

7Question

Necrosis pattern — liquefactive?

Réponse

Tissue digested into pus, common in brain.

8Question

Necrosis pattern — caseous?

Réponse

Cheeselike, granuloma-like, in tuberculosis.

9Question

Necrosis pattern — fat?

Réponse

Chalky deposits from pancreatic lipase activity.

10Question

Necrosis pattern — fibrinoid?

Réponse

Bright pink immune complex deposits in vessel walls.

11Question

Apoptosis — morphology?

Réponse

Cell shrinkage, chromatin condensation, apoptotic bodies.

12Question

Apoptosis — pathways?

Réponse

Mitochondrial (BAX/BAK) and extrinsic (Fas/FADD).

13Question

Autophagy — process?

Réponse

Sequestration of cytoplasm into autophagosomes for degradation.

14Question

Necroptosis — mechanism?

Réponse

RIPK1/RIPK3/MLKL pathway causing membrane rupture.

15Question

Oxidative stress — cause?

Réponse

Excess ROS damaging lipids, proteins, DNA.

16Question

Lipid peroxidation — effect?

Réponse

Membrane damage via free radicals attacking fatty acids.

17Question

Hypoxia — definition?

Réponse

Reduced oxygen supply to tissues.

18Question

Reperfusion injury — paradox?

Réponse

Restored blood flow causes further cell damage.

19Question

Chemical injury — example?

Réponse

Mercuric chloride binding to sulfhydryl groups.

20Question

Infectious injury — mechanism?

Réponse

Pathogens damage cells directly or via immune response.

21Question

Immunologic injury — example?

Réponse

Complement activation and immune complex deposition.

22Question

Cellular adaptation — types?

Réponse

Hypertrophy, hyperplasia, atrophy, metaplasia.

23Question

Intracellular pigment — example?

Réponse

Lipofuscin, hemosiderin, carbon (anthracosis).

24Question

Calcification — types?

Réponse

Dystrophic (injury sites) and metastatic (hypercalcemia).

Teste-toi avec le QCM

Teste tes connaissances avec un QCM de 24 questions sur Cell Injury and Death Mechanisms.

1. What is the best term for a reversible change in cell structure and function that helps a cell survive a stress when adaptive limits are not exceeded?

2. Which sequence best describes what happens when a stress is removed before damage becomes severe?

Faire le QCM →

Consultez la fiche

Révisez le cours complet dans la fiche de révision de Cell Injury and Death Mechanisms.

Voir la fiche →

Cours similaires

Crée tes propres flashcards

Importe ton cours et l'IA génère des flashcards en 30 secondes.

Générateur de flashcards